Search results for "L-type calcium channel"

showing 5 items of 5 documents

L-type calcium channel activity in human atrial myocytes as influenced by 5-HT.

1993

5-Hydroxytryptamine (10 mumol/l; 5-HT) exerted a positive inotropic effect associated with an increase in the Ca2+ current (ICa) in the human right atrium. For detailed analysis, L-type Ca2+ channel currents were recorded from cell-attached patches using 100 mmol/l Ba2+ as charge carrier. Ca2+ channel activity was identified, first, by burst-like inwardly directed currents and, second, by the appearance of long channel openings promoted by Bay K 8644 (1 mumol/l) upon repetitive depolarizations from -80 to 0 mV. The unitary conductance of the Ca2+ channel amounted to 25.8 pS. During superfusion with 5-HT, ensemble averaged (mean) current was enhanced by about 60%. The increase in mean curren…

MaleSerotoninchemistry.chemical_elementAction PotentialsGatingCalciumIn Vitro TechniquesMoleCyclic AMPHumansL-type calcium channelPhosphorylation5-HT receptorAgedPharmacologyMyocardiumIsoproterenolInfantDepolarizationHeartGeneral MedicineAnatomy3-Pyridinecarboxylic acid 14-dihydro-26-dimethyl-5-nitro-4-(2-(trifluoromethyl)phenyl)- Methyl esterMiddle AgedMyocardial ContractionElectrophysiologyElectrophysiologyKineticschemistryChild PreschoolBiophysicsFemaleCalcium ChannelsIon Channel GatingCommunication channelNaunyn-Schmiedeberg's archives of pharmacology
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Capsaicin differentially modulates voltage-activated calcium channel currents in dorsal root ganglion neurones of rats

2005

It is discussed whether capsaicin, an agonist of the pain mediating TRPV1 receptor, decreases or increases voltage-activated calcium channel (VACC) currents (ICa(V)). ICa(V) were isolated in cultured dorsal root ganglion (DRG) neurones of rats using the whole cell patch clamp method and Ba 2+ as charge carrier. In large diameter neurones (>35Am), a concentration of 50AM was needed to reduce ICa(V) (activated by depolarizations to 0 mV) by 80%, while in small diameter neurones (30Am), the IC50 was 0.36 AM. This effect was concentration dependent with a threshold below 0.025 AM and maximal blockade (>80%) at 5AM. The current–voltage relation was shifted to the hyperpolarized direction with an…

Pain ThresholdPatch-Clamp TechniquesTRPV1TRPV Cation ChannelsN-type calcium channelSecond Messenger SystemsMembrane PotentialsGanglia SpinalAnimalsL-type calcium channelPatch clampRats WistarMolecular BiologyCell SizeNeuronsDose-Response Relationship DrugVoltage-dependent calcium channelChemistryGeneral NeuroscienceCalcium channelT-type calcium channelCalciseptineAnatomyRatsRats Inbred LewBiophysicsCalcium ChannelsNeurology (clinical)CapsaicinSignal TransductionDevelopmental BiologyBrain Research
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Kinetics and state-dependent effects of verapamil on cardiac L-type calcium channels.

1996

The voltage dependence and the kinetics of block by verapamil of L-type calcium current (ICa) were investigated in ventricular myocytes from rat hearts using the whole-cell patch-clamp technique. ICa was elicited repetitively in response to depolarizing voltage pulses from -80 mV to 0 mV at different pulse intervals and durations. Verapamil reduced the magnitude of ICa in a frequency-dependent manner without tonic component. The time course of ICa remained unchanged suggesting that not open but inactivated channels were affected by the drug. The interaction of verapamil with inactivated channels was investigated by the application of twin pulses. In the presence of verapamil, the duration o…

PharmacologyVoltage-dependent calcium channelChemistryKineticsTime constantAnalytical chemistryDepolarizationHeartGeneral MedicineIn Vitro TechniquesCalcium Channel BlockersRatsKineticsDrug concentrationNuclear magnetic resonanceVerapamilState dependentBariummedicineVerapamilAnimalsL-type calcium channelCalcium Channelsmedicine.drugNaunyn-Schmiedeberg's archives of pharmacology
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Differential blockade by nifedipine and ω-conotoxin GVIA of α1- and β1-adrenoceptor-controlled calcium channels on motor nerve terminals of the rat

1990

Electrically evoked release of [3H]acetylcholine ([3H]ACh) from the rat phrenic nerve and its facilitation by stimulation of presynaptic alpha 1- and beta 1-adrenoceptors were investigated in the absence and presence of nifedipine and omega-conotoxin GVIA. Both calcium channel antagonists did not modify electrically evoked [3H]ACh release, but selectively blocked the effect triggered by both facilitatory adrenergic receptors. The increase in [3H]ACh release mediated via beta 1-adrenoceptor activation was abolished by low concentrations (1 nM) of omega-conotoxin GVIA, whereas nifedipine (100 nM) abolished the facilitatory effect mediated via alpha 1-adrenoceptor stimulation. Therefore, the b…

medicine.medical_specialtyNifedipineNeuromuscular JunctionMollusk Venomschemistry.chemical_elementIn Vitro TechniquesCalciumN-type calcium channelMotor Endplatecomplex mixturesNeuromuscular junctionNifedipineomega-Conotoxin GVIAInternal medicinemedicineAnimalsL-type calcium channelVoltage-dependent calcium channelChemistryGeneral NeuroscienceCalcium channelRats Inbred StrainsCalcium Channel BlockersAcetylcholineElectric StimulationRatsReceptors AdrenergicPhrenic Nervemedicine.anatomical_structureEndocrinologyCalcium ChannelsAcetylcholinemedicine.drugNeuroscience Letters
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Vascular effects of progesterone: Role of cellular calcium regulation

2001

Abstract —Vascular actions of progesterone have been reported, independently of estrogen, affecting both blood pressure and other aspects of the cardiovascular system. To study possible mechanisms underlying these effects, we examined the effects of P in vivo in intact rats and in vitro in isolated artery and vascular smooth muscle cell preparations. In anesthetized Sprague-Dawley rats , bolus intravenous injections of P (100 μg/kg) significantly decreased pressor responses to norepinephrine (0.3 μg/kg). In vitro, progesterone (10 −8 to 10 −5 mmol/L) produced a significant, dose-dependent relaxation of isolated helical strips, both of rat tail artery precontracted with KCl (60 mmol/L) or a…

medicine.medical_specialtyVasopressinVascular smooth musclemedicine.drug_classchemistry.chemical_elementBiologyCalciumCalcium in biologyNorepinephrine (medication)EndocrinologychemistryEstrogenInternal medicineCirculatory systemInternal MedicinemedicineL-type calcium channelmedicine.drug
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